PATHOLOGY № 5TASK №o1A 24-year-old patient was admitted to a psychiatric hospitalwith a diagnosis of Hysterical Neurosis. He had complains ofheadaches, irritability, nausea, poor sleep. During conversationwith a doctor the patient demonstrated groans, sobs, the urge tovomit, the presentation of unsubstantiated claims to medicalstaff. On examination: signs of somatic and neurologicalpathology were not detected. A day later, during a conversationwith a doctor, the patient had an emotional “explosion" (shouting,crying, moans, chaotic movements), followed by a short loss ofconsciousness and the development of hysterical paralysis of theleft hand.Questions:6. What disorders or sensory sensitivity can develop in thisdisease?7. What diseases risk factor is neurosis?

6. Hysterical Neurosis: Neurotic disorder distinguished by violent emotional outbursts and…

Answered: PATHOLOGY № 5 TASK No 1 A 24-year-old…

Essentials of Pharmacology for Health Professions

7th Edition

ISBN:9781305441620

Author:WOODROW

Publisher:WOODROW

Chapter22: Anticonvulsants, Antiparkinsonian Drugs, And Agents For Alzheimer’s Disease

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A 24-year-old patient was admitted to a psychiatric hospital with a diagnosis of Hysterical Neurosis. He had complains of headaches, irritability, nausea, poor sleep. During conversation with a doctor the patient demonstrated groans, sobs, the urge to vomit, the presentation of unsubstantiated claims to medical staff. On examination: signs of somatic and neurological pathology were not detected. A day later, during a conversation with a doctor, the patient had an emotional "explosion" (shouting, crying, moans, chaotic movements), followed by a short loss of consciousness and the development of hysterical paralysis of the left hand. Questions: 6. What disorders of sensory sensitivity can develop in this disease? 7. What diseases risk factor is neurosis?
A 24-year-old patient was admitted to a psychiatric hospital with a diagnosis of Hysterical Neurosis. He had complains of headaches, irritability, nausea, poor sleep. During conversation with a doctor the patient demonstrated groans, sobs, the urge to vomit, the presentation of unsubstantiated claims to medical staff. On examination: signs of somatic and neurological pathology were not detected. A day later, during a conversation with a doctor, the patient had an emotional "explosion" (shouting, crying, moans, chaotic movements), followed by a short loss of consciousness and the development of hysterical paralysis of the left hand. Questions: 4. What locomotor (except paralysis) disorders are possible with this pathology. 5. List the typical vegetative disorders in this pathology.
A 24-year-old patient was admitted to a psychiatric hospital with a diagnosis of Hysterical Neurosis. He had complains of headaches, irritability, nausea, poor sleep. During conversation with a doctor the patient demonstrated groans, sobs, the urge to vomit, the presentation of unsubstantiated claims to medical staff. On examination: signs of somatic and neurological pathology were not detected. A day later, during a conversation with a doctor, the patient had an emotional "explosion" (shouting, crying, moans, chaotic movements), followed by a short loss of consciousness and the development of hysterical paralysis of the left hand. Questions: 1. List the signs of hysterical neurosis in a patient. 2. Describe the etiology of the disease. 3. Describe its pathogenesis
Intermittent Claudications evoke :- a- visceral painb- deep painc- cutaneous hyperalgesiad- colicky pain
A 65 year old man is brought to the emergency department after an episode of a transient ische
The patient is a 9-year old male, grade 2 student, managed as a case of Paraplegia sec to Spinal Cord Injury ASIA A (anatomic level: T10, sensory level: T10, motor level: T10, neurologic level: T10). Pertinent PE findings: Tightness B heelcordsHyporeflexive both LE, Hypotonic both LEsWeakness on B LEMyotomes- bilateral C5-T1 level MMT: 5/5, bilateral L2-S1 level MMT: 0/5Sensory deficit on B LEDermatomes- bilateral C2-T10 level 2/2, bilateral T11-S3 level 0/2Lax sphincter tone, (-) bladder filling sensationLimited Activity: moderate assist in bed mobility, transitions, transfers, moderate assist in ADLs What are two things that you will consider when handling and transitioning a paraplegic patient?
The patient is a 9-year old male, grade 2 student, managed as a case of Paraplegia sec to Spinal Cord Injury ASIA A (anatomic level: T10, sensory level: T10, motor level: T10, neurologic level: T10). Pertinent PE findings: Tightness B heelcordsHyporeflexive both LE, Hypotonic both LEsWeakness on B LEMyotomes- bilateral C5-T1 level MMT: 5/5, bilateral L2-S1 level MMT: 0/5Sensory deficit on B LEDermatomes- bilateral C2-T10 level 2/2, bilateral T11-S3 level 0/2Lax sphincter tone, (-) bladder filling sensationLimited Activity: moderate assist in bed mobility, transitions, transfers, moderate assist in ADLs What are two recommendations for this patient in order to minimize potential pressure sores from prolonged sitting?
The Alaska Department of Public Health was notified that foodborne illness had occurred in fishermen aboard a fishing boat off the Alaska peninsula. The fishermen had eaten steamed clams and mussels, boiled rice, boiled potatoes, and green salad. No alcohol was consumed. Symptoms experienced by the fishermen included numbness of the lips, tingling of the extremities, uncoordinated movements, incoherent speech, and nausea. Identify the etiologic (causative) agent of this outbreak of food poisoning. How did the food get contaminated, and what item was contaminated? What is the treatment, if any? How could this illness have been prevented?
Describe the clinical presentation and laboratory investigation of DBA and explain the typical laboratory findings associated with this condition. Page 5 of 7 1759 words LE 3 Focus + 1242 MacB
. A 19-year-old male with known generalized epilepsy was brought to the emergency department for convulsive status epileptics. He has had nausea, emesis, and a low grade fever for the last 5 days and has been unable to take his home antiepileptic drug. He is having ongoing low amplitude, rhythmic clonic movements of his bilateral arms and legs. His vitals are as follows: heart rate 86, blood pressure 106/68, SpO, 100% on 2 L nasal canula, temperature 100.2°C. EMS administered 2 mg of lorazepam and had cessation of clonic movements but still altered and not back to baseline mental state. What is the next best medication treatment for this patient? A. Intubate the patient and start propofol B. Monitor the patients for few hours and order EEG C. Additional lorazepam to dose of 0.1 mg/kg followed by fosphenytoin with loading dose of 15 mg/kg IV D. Obtain CT head to further evaluate the etiology and rule out structural abnormalities
What is status epilepticus? And what are the management options for this? Please shortly answer at your own easy words. Answer should be to the point.
Episode 1 Mary, a 44-year-old mother of two, experiencing a prolonged episode of numbness in her chin and lower lip. Two days prior to her appointment, she felt a prickling sensation like “pins and needles” at the right corner of her mouth. The sensation extended to her lower lip and chin. The examination revealed only a superficial hypoesthesia of the chin and lower lip (numb chin syndrome). CT scan of the affected region showed no abnormalities in the jaw, neck, or pharynx. The numbness and hypoesthesia spontaneously disappeared gradually over a few weeks. Episode 2 Four months later, Mary felt a stabbing pain in her upper jaw and teeth that radiated out to the side of her nose. Over the next several days, she experienced several more episodes of this intense pain. A visit to the dentist revealed no abnormalities and she was referred to her physician for an evaluation. Her physician scheduled an appointment for a complete neurological exam the following week. Episode 3 Three nights…

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