Release of trypsin and chymotrypsin in their active form can result in the digestion of the pancreas or small intestine itself. What mechanism does the body employ to prevent its self-destruction?
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- Release of trypsin and chymotrypsin in their active form can result in the digestion of the pancreas or small intestine itself. What mechanism does the body employ to prevent its self-destruction?
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- How does a ketogenic diet decrease the level of urea in obese patients and increase the level of creatine in obese patients? (explain on a molecular level)Look up trypsin and answer the following questions? a) Where in the body is this made? b) What type of enzyme is it?Indicate whether each of the following statements is true or false. If false, correct the statement or provide a brief explanation for why it is a) The presence of lipids in the diet slows down digestion in the b) Insulin increases the rate of glucose uptake by the c) Glucagon is the hormone that signals low blood glucose d) Chylomicrons carry TAGs from the diet into the e) During fasting and starvation the liver produces ketone bodies which fuel the heart and f) The muscles can use glucose, fatty acids and ketone bodies for g) When glucose enters the cell it is immediately and reversibly h) Epinephrine stimulates breakdown of glycogen in muscles when there is an immediate need for energy by muscle i) Glucagon stimulates glycogenolysis to maintain the blood glucose j) With high [carbohydrate] levels, excess glucose (after glycogen storage has reached a maximum) is converted to fat, leading to high [malonyl-CoA]. k) Malonyl-CoA inhibits carnitine acyltransferase II , thus slowing…
- Leptin inhibits eating and is secreted in amounts in direct proportion to body fat. Moreover, obese people have normal amounts of leptin and leptin receptor. Why, then, do people become obese?The most common form of malnutrition in children in the world, kwashiorkor, is caused by a diet having ample calories but little protein. The high levels of carbohydrate result in high levels of insulin. What is the effect of high levels of insulin on (a) lipid utilization? (b) protein metabolism? (c) Children suffering from kwashiorkor often have large distended bellies caused by water from the blood leaking into extracellular spaces. Suggest a biochemical basis for this condition.One consequence of starvation is a reduction in muscle mass. What happens to the muscle proteins?
- Why is daily ingestion of vitamin C more critical than daily ingestion of vitamin A?Which of the following statements is true about brain metabolism in starvation? a) The brain can use glucogenic amino acids for energy b) The brain can only use glucose as fuel c) Up to a quarter of energy requirement of the brain can come from fatty acids d) Up to a half of energy requirement of the brain can come from ketone bodiesDecide whether each of the following statements is true or false. If false, explain why.(a) The amino acid pool is found mainly in the liver.(b) Nitrogen-containing compounds can be stored in fatty tissue.(c) Some hormones and neurotransmitters are synthesized from amino acids.
- In healthy adults, the concentration of glucose in blood is approximately80 to 110 milligrams per deciliter (mg/dl). After a carbohydrate-richmeal, however, the concentration may spike to 140 mg/dl. Describe thehormonal action that returns blood glucose to normal.Glucagon facilitates the adaptation of our body to fasting. Which enzymes of glucides (carbohydrates) metabolism are regulated, directly or undirectly, in response to the secretion of glucagon by pancreas?.e) (i) People living in tropical region usually do not have deficiency in vitamin D even if their diet is low in vitamin D. Briefly explain this. (ii) Briefly describe the activation of vitamin D.